Tumeric isn't expensive, isn't considered harmful (if you have kidney stones it might be though), and as discussed before, there are many scientific clinical trials regarding potential of Tumeric.
This is an excerpt of an article at a natural website, but it was linked to by Sloan Kettering.
In vitro studies suggest that curcumin acts as a weak phytoestrogen (43), exhibits neuroprotective (42), antiproliferative and preventative effects against cancer (3) (4) (5) (6) (7) (8) (35). Furthermore, curcumin induces apoptosis in human colon cancer (14), promyelocytic leukemia cells (15), and inhibits growth of uterine leiomyosarcoma cells (45). Curcumin also potentiated gemcitabine action in both in vitro and in vivo studies of pancreatic cancer (17). In a phase II trial in pancreatic cancer patients, down-regulation of NF-kappa B and cyclooxygenase-2 were observed (29).
Oral administration is well tolerated, but bioavailability is relatively low (1) (2) (29). Following absorption, curcuminoids are rapidly metabolized. But a recent study in rats shows that bioavailability of curcumin can be increased when coadministered with piperine (38).
Animal studies indicate that curcumin may inhibit cyclophosphamide in treating breast cancer (16), but results from a recent, phase I trial found a combination of curcumin and docetaxel to be safe (36). More research is necessary, but it is advisable for cancer patients undergoing chemotherapy to limit intake of turmeric. Patients with gastrointestinal disorders or those predisposed to kidney stone formation (13) should use this supplement with caution.
Mechanism of Action
Turmeric has anti-inflammatory and choleretic actions. Both curcuminoids (curcumin) and volatile oils are responsible for the anti-inflammatory activity, which may be due to leukotriene inhibition. Curcuminoids induce glutathione S-transferase and are potent inhibitors of cytochrome P450. Turmeric acts as a free radical scavenger and antioxidant, inhibiting lipid peroxidation
and oxidative DNA damage. It also inhibits activation of NF-kB 17, 20, c-jun/AP-1 function, and activation of the c-Jun NH2-terminal kinase (JNK) pathway.
In vitro and animal models of breast cancer show that turmeric may inhibit chemotherapy-induced apoptosis via inhibition of the JNK pathway and generation of reactive oxygen species (ROS). Studies also suggest that curcumin induces apoptosis in human colon cancer cells independent of p21 expression (14). In addition, in vitro and in vivo studies report that NF-kB-mediated resistance of cancer cells to gemcitabine and ɣ-radiation was repressed by curcumin administration (17) (21). In laboratory tests, curcumin's antitumor actions appear to be due to interactions with arachidonate metabolism and its in vivo antiangiogenic properties (16) (22). Another possible chemopreventive mechanism of curcumin maybe via binding and activating the vitamin D receptor (VDR), thereby protecting tissues such as small intestine and colon where VDRs are expressed and vitamin D is known to serve anticancer function (30). Curcumin inhibited uterine leiomyosarcoma cells' growth by targeting the AKT-mTOR pathway for inhibition (45).